1
Ic immunoprecipitation and input DNA. H3K9, Lysine 9 of histone H3; H4K8, Lysine 8 of histone H4; H3K4, Lysine 4 of histone H3; H4K20, Lysine 20 of histone H4; ac, acetylated; me, monomethylated; me2, dimethylated; me3, trimethylated. The significant p-values are shown.Schwarzenbach et al. BMC Cancer 2014, 14:796 http://www.biomedcentral.com/1471-2407/14/Page 10 ofTherefore, the predominant occurr
1
Homologs: implications regarding origin of eukaryotic cells and of endoplasmic reticulum. Proc. Natl. Acad. Sci. USA 91:2895?899. 103. Gupta, R. S., K. Bustard, M. Falah, and D. Singh. 1997. Sequencing of heat shock protein 70 (DnaK) homologs from Deinococcus proteolyticus and Thermomicrobium roseum and their integration in a protein-based phylogeny of prokaryotes. J. Bacteriol. 179:345?57. 104. G
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Rotein TCP-1 and the `chaperonin' family of bacterial (GroEL, 60-65 kDa heat shock antigen) and eukaryotic proteins. Biochem. Int. 20:833?41. 96. Gupta, R. S. 1995. Evolution of the chaperonin families (Hsp60, Hsp10 and Tcp-1) of proteins and the origin of eukaryotic cells. Mol. Microbiol. 15: 1?1. 97. Gupta, R. S. 1995. Phylogenetic analysis of the 90 kD heat shock family of protein sequences and
1
Ska), extracellular protein factor ( epf), or protein F2 ( prtF2) (B) were tested for surface PK/FXIIa activity after incubation with normal human plasma. (C) The PK/FXIIa activity at the surfaces of M49 wild-type and ska mutant strains was examined after exposure to normal or plasminogen-deficient plasma. Kaolin served as a positive control for plasmin-independent contact system activation. (D) P
1
Of a part of the type 1 LacNAc (Gal -1,3-GlcNAc) and type 2 LacNAc (Gal -1,4-GlcNAc) structures that build the scaffold for blood group H and Lewis-type units (58). Some human pathogens use the GlcNAc residue as a binding receptor; e.g., the fimbrial adhesin F17-G of enterotoxigenic E. coli binds to N-acetylglucosamine-presenting receptors on the microvilli of the intestinal epithelium of ruminant
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The latest. Figure 60 shows a significant increase in admissions among the elder groups along the five-year periods. The severity scores increased significantly as shown in Figure 61 (p
1
E results indicate that secreted SK is a potent contact system activator, exhibiting its function independently from a contact surface. The chromogenic substrate S-2302 is specific for PK/FXIIa but is also sensitive to plasmin. We therefore measured the PK/FXIIa activity after addition of chloromethyl-ketone D-Val-Phe-Lys, which is an efficient inhibitor of plasmin (30) and the SK-plasminogen comp
1
Rotein TCP-1 and the `chaperonin' family of bacterial (GroEL, 60-65 kDa heat shock antigen) and eukaryotic proteins. Biochem. Int. 20:833?41. 96. Gupta, R. S. 1995. Evolution of the chaperonin families (Hsp60, Hsp10 and Tcp-1) of proteins and the origin of eukaryotic cells. Mol. Microbiol. 15: 1?1. 97. Gupta, R. S. 1995. Phylogenetic analysis of the 90 kD heat shock family of protein sequences and
1
In this research, the effect regarding roxithromycin (ROX) on unstable essential fatty acid (VFA) recuperation coming from WAS anaerobic fermentation had been looked into. The particular trial and error results showed #links# in which ROX developed a good factor on the creation of VFAs. With all the improve of ROX dosages from Zero to 100?mg/kg TSS, the maximum build up regarding VFAs increased fr
1
Noma in situ; DC-SCRIPT, dendritic cell-specific transcript gene; ERBB2+, HER2neu-positive; ESR, estrogen receptor gene; IDC, infiltrating ductal carcinoma; ILC, infiltrating lobular carcinoma; PGR, progesterone receptor gene; pT1, small tumor without lymphatic/vascular invasion.(Spearman's rho = 0.87; P
1
Ith DFS, MFS, OS, and PFS, respectively. In multivariable analysis, Cox proportional hazards models for DFS, MFS, OS, and PFS were applied to test DCSCRIPT levels added to models with traditional factors. The proportional hazards assumptions were checked with Schoenfeld residuals. The analyses were stratified if necessary. The models for DFS, MFS, and OS for LNN patients who had not received adjuv
1
Ic immunoprecipitation and input DNA. H3K9, Lysine 9 of histone H3; H4K8, Lysine 8 of histone H4; H3K4, Lysine 4 of histone H3; H4K20, Lysine 20 of histone H4; ac, acetylated; me, monomethylated; me2, dimethylated; me3, trimethylated. The significant p-values are shown.Schwarzenbach et al. BMC Cancer 2014, 14:796 http://www.biomedcentral.com/1471-2407/14/Page 10 ofTherefore, the predominant occurr
1
Tcome are listed in Table 1.RNA isolation and quantitative RT-PCRThe protocol to study biological markers associated with disease outcome was approved by the medical ethics committee of the Erasmus Medical Center (Rotterdam, The Netherlands) (MEC 02.953). This retrospective study used 1,505 M0 (no metastasis) and 32 M1 (with metastasis) blind-coded freshly frozen primary tumor tissues of female pa
1
Our intensive care unit, 131 of them, were 80 years ( 8,8 ) with a mean ageA946 Evolution of the number of admissions, age, scales of severity, length of stay and mortality in a general intensive care unit of a university hospital over 15 years L. Martinez Pujol, R. Algarte Dolset, B. S chez Gonz ez, S. Quintana Riera, J. Trenado varez Hospital Universitari Mutua Terrassa, Intensive Care Medici
1
Rotein TCP-1 and the `chaperonin' family of bacterial (GroEL, 60-65 kDa heat shock antigen) and eukaryotic proteins. Biochem. Int. 20:833?41. 96. Gupta, R. S. 1995. Evolution of the chaperonin families (Hsp60, Hsp10 and Tcp-1) of proteins and the origin of eukaryotic cells. Mol. Microbiol. 15: 1?1. 97. Gupta, R. S. 1995. Phylogenetic analysis of the 90 kD heat shock family of protein sequences and
1
Romogenic substrate (S-2302), specific for PK and FXIIa, was used to measure contact activation. Both strains exhibited PK/FXIIa activity on their surfaces (Fig. 1A). Interestingly, compared to the wild types, knockout of the M protein in these strains did not change PK/FXIIa activity significantly (Fig. 1A), suggesting that activation of contact system factors occurs independently of the M protei
1
Our intensive care unit, 131 of them, were 80 years ( 8,8 ) with a mean ageA946 Evolution of the number of admissions, age, scales of severity, length of stay and mortality in a general intensive care unit of a university hospital over 15 years L. Martinez Pujol, R. Algarte Dolset, B. S chez Gonz ez, S. Quintana Riera, J. Trenado varez Hospital Universitari Mutua Terrassa, Intensive Care Medici
1
Ptide. Proteolytic activities are indicated with green arrows, and steps inhibited by C1-INH are shown by red bars. From Zuraw [1]. ?2008 Massachusetts Medical Society. Reprinted with permission from Massachusetts Medical Society [1]Recombinant Human C1 Esterase Inhibitorpathway due to insufficient levels of functional C1-INH results in unregulated cleavage of high molecular weight kininogen by ka
1
Ic immunoprecipitation and input DNA. H3K9, Lysine 9 of histone H3; H4K8, Lysine 8 of histone H4; H3K4, Lysine 4 of histone H3; H4K20, Lysine 20 of histone H4; ac, acetylated; me, monomethylated; me2, dimethylated; me3, trimethylated. The significant p-values are shown.Schwarzenbach et al. BMC Cancer 2014, 14:796 http://www.biomedcentral.com/1471-2407/14/Page 10 ofTherefore, the predominant occurr
1
E was 15 cases in 2013 compared to 11 in 2014. The strains with resistance or intermediate sensitivity to IMP were 5/15 in 2013 (33 ), while it was only 1/11 (9 ) in 2014. First samples of MRSA, in 2013, were 3, whereas there was only one in 2014. Two cases of Klebsiella pneumoniae ESBL were identified in 2013 and 3 cases in 2014. There was one case of Clostridium difficile in 2013 and none in 2
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Ds Enzymol. 266:418?27. 62. Felsenstein, J. 1997. Cases in which parsimony and compatibility methods will be positively misleading. Syst. Zool. 27:401?10. 63. Feng, D. F., G. Cho, and R. F. Doolittle. 1997. Determining divergence times with a protein clock: update and reevaluation. Proc. Natl. Acad. Sci. USA 94:13028?3033. 64. Fitch, W. M. 1997. Toward defining the course of evolution: minimum cha
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E results indicate that secreted SK is a potent contact system activator, exhibiting its function independently from a contact surface. The chromogenic substrate S-2302 is specific for PK/FXIIa but is also sensitive to plasmin. We therefore measured the PK/FXIIa activity after addition of chloromethyl-ketone D-Val-Phe-Lys, which is an efficient inhibitor of plasmin (30) and the SK-plasminogen comp
1
Uctal carcinoma in situ (DCIS) component or infiltrating lobular carcinoma compared with infiltrating ductal carcinomas (both P
1
Urse of the disease without the confounding effect of systemic adjuvant therapy), we restricted our next analyses of MFS to those 837 LNN disease patients who had not received (neo)adjuvant systemic therapy. The significant relationships of DC-SCRIPT as a continuous variable in these univariate analyses justified the use of the previously identified cut point that dichotomized the cohort in 33.3
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An DD, Ho V, Meibohm B, et al. The challenge of indication extrapolation for infliximab biosimilars. Biologicals. 2014;42(4):177?3. Weise M, Kurki P, Wolff-Holz E, Bielsky MC, Schneider CK. Biosimilars: the science of extrapolation. Blood. 2014;124(22): 3191?. Ebbers HC. Biosimilars: in support of extrapolation of indications. J Crohns Colitis. 2014;8(5):431?. Liu J, et al. Assessing analytical si
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